However, testosterone in physiologic doses could even be helpful in patients with angina , have higher ischemic thresholds and improved quality of life . Short-term intracoronary administration of testosterone induces coronary artery dilation and will increase coronary blood circulate in males with established coronary artery disease . Therefore, an interaction of endogenous elements of the patient’s threat profile as properly as the testosterone dosing could also be issues of whether or not antagonistic occasions occur. Erythrocytosis has been reported as a potential impact of testosterone, which may be beneficial in patients with anemia. The greatest threat of erythrocytosis occurs with injectable forms of testosterone compared with transdermal preparations, particularly in supraphysiologic doses. An improve of up to 44% of the baseline hematocrit has been reported .

• Short-term intracoronary administration of testosterone induces coronary artery dilation and will increase coronary blood circulate in men with established coronary artery illness .
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Unfortunately, the information on using excessive dose anabolic steroids in humans are mainly supplied as case reports or small studies that lack sufficient management groups. In addition, the data out there within the literature does not account for the steroid kind or dose, as neither may be known. Patients additionally could also be taking concomitant stimulants, corresponding to ephedra, which may confound data and the potential for antagonistic events. It is most likely going that the frequency of cardiac occasions is underreported in the medical literature. Supraphysiologic doses of anabolic-androgenic steroids , which embody more than 30 natural and synthetic derivatives of testosterone, may trigger a large spectrum of medical sequalae.

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We report a case of an acute ST-segment elevation myocardial infarction in a patient with conventional cardiac risk components using supraphysiologic doses of supplemental, intramuscular testosterone. In addition, this patient also had polycythemia, doubtless secondary to high-dose testosterone. The patient underwent profitable percutaneous intervention of the best coronary artery. Physicians and patients ought to concentrate on the medical penalties of anabolic steroid abuse. Development of an atheromatous plaque perpetuates endothelial dysfunction and promotes platelet aggregation and intracoronary thrombus formation (Ajayi et al 1995; Nieminen et al 1996). Moreover, a element of endothelial dysfunction has been proposed, which may contribute to irregular vessel reactivity.

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Moreover, testosterone has been used in the therapy of reduced muscle mass and bone density as well as anemia. The function testosterone may play in the growth of cardiovascular disease has more and more turn out to be the topic of present curiosity . Anabolic androgenic steroid use has elevated over the past decade, regardless of its potential for antagonistic effects. They proceed to be used by Olympic degree athletes, and have even spread to highschool degree athletes, noncompetitive bodybuilders and recreational athletes (Sullivan et al 1998; Dhar et al 2007).

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